Key result
Monensin inhibits in vitro SARS-CoV-2 replication at ~11 µM, highlighting synergy with cholesterol-targeting compounds.
Why the study?
Most of the around 200 repurposed drug candidates for COVID-19 showed moderate anti-SARS-CoV-2 activity, but gaining insights into their mechanisms of action was needed to better understand infection and develop therapeutics.
Drugs co-targeting cholesterol homeostasis and microtubule cytoskeleton organization show potential synergistic effects with antivirals against SARS-CoV-2.
May support preclinical testing of monensin; leaves open whether cholesterol-microtubule targets enhance antivirals in vivo.
The global efforts in the past few months have led to the discovery of around 200 drug repurposing candidates for COVID-19. Although most of them only exhibited moderate anti- SARS-CoV-2 activity, gaining more insights into their mechanisms of action could facilitate a better understanding of infection and the development of therapeutics. Leveraging large-scale drug-induced gene expression profiles, we found 36% of the active compounds regulate genes related to cholesterol homeostasis and microtubule cytoskeleton organization. The expression change upon drug treatment was further experimentally confirmed in human lung primary small airway. Following bioinformatics analysis on COVID-19 patient data revealed that these genes are associated with COVID-19 patient severity. The expression level of these genes also has predicted power on anti-SARS-CoV-2 efficacy in vitro, which led to the discovery of monensin as an inhibitor of SARS-CoV-2 replication in Vero-E6 cells. The final survey of recent drug- combination data indicated that drugs co-targeting cholesterol homeostasis and microtubule cytoskeleton organization processes more likely present a synergistic effect with antivirals. Therefore, potential therapeutics should be centered around combinations of targeting these processes and viral proteins.
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Xing et al. (2021) studied COVID-19 (SARS-CoV-2 infection). Monensin vs. Control (DMSO) was evaluated on Inhibition of SARS-CoV-2 replication in Vero-E6 cells (IC50) (IC50 11 µM). Monensin inhibited SARS-CoV-2 replication in vitro with an IC50 of 11 µM, supporting the finding that compounds targeting cholesterol homeostasis and microtubule organization synergize with antivirals.
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