Key result
Defective sarcoplasmic reticulum-mitochondrial crosstalk increases arrhythmic risk in cardiac disease.
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Design
Review
Impaired SR-mitochondria crosstalk may increase arrhythmic risk; leaves open mechanistic targets for arrhythmia prevention pending validation.
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Hamilton et al. (2021) studied this question. Defects in crosstalk between the sarcoplasmic reticulum and mitochondria increase arrhythmic risk in cardiac disease.
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