Key result
Environmental tobacco smoke exposure is linked to increased CHD risk in non-smokers.
Why the study?
Does environmental tobacco smoke (ETS) exposure increase the risk of coronary heart disease in non-smokers?
Does environmental tobacco smoke (ETS) exposure increase the risk of coronary heart disease in non-smokers?
Passive smoking is associated with an increased risk of coronary heart disease, highlighting the cardiovascular benefits of smoking cessation for both smokers and those exposed to secondhand smoke.
Answers are grounded in this paper and cite their sources.
Passive smoking exposure may elevate CHD risk; extends observational data but leaves causal effects and practice change open.
A large series of clinico-epidemiological studies, meta-analyses and experimental findings have concluded that there is a relationship between coronary heart disease (CHD) and passive smoking either after acute or chronic exposure. Cigarette smoking is the most important cause of premature death in industrialized countries because it is associated with an increased risk of developing several types of cancer and arterial disease. In family homes as well as in workplaces, environmental tobacco smoke (ETS) exposure is associated with an increased risk of CHD in exposed non-smokers when compared to un-exposed non-smokers. Different anatomical structures are damaged by ETS. The endothelium, artery wall and heart are target organs for passive smoking. Therefore, smoking cessation will benefit both smokers and those exposed to ETS.
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Leone et al. (2004) conducted a review in Coronary heart disease. Environmental tobacco smoke (ETS) exposure vs. Un-exposed non-smokers was evaluated on Coronary heart disease. Environmental tobacco smoke exposure is associated with an increased risk of coronary heart disease in exposed non-smokers when compared to un-exposed non-smokers.
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