Why the study?
Functional TR and RV remodeling form a vicious cycle linked to adverse outcomes, creating a need for therapies that reverse RV remodeling and reduce TR.
Does empagliflozin, sustained-release carvedilol, or their combination improve right ventricular remodeling and reduce tricuspid regurgitation severity in patients with significant functional tricuspid regurgitation?
Population
56 patients with significant functional TR
Comparison
Carvedilol SR plus empagliflozin vs carvedilol SR vs empagliflozin vs placebo
Design
Randomized trial with a 2 × 2 factorial design
Follow-up
12 months
Key result
Empagliflozin did not significantly reduce right ventricular end-systolic volume index compared with placebo (MD 1.67 mL/m2, p=0.708), although it significantly reduced secondary measures of tricuspid regurgitation severity.
Authors
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Empagliflozin should not target right ventricular remodeling in functional tricuspid regurgitation; extends evidence for possible regurgitation reduction pending larger trials.
RCT (n=56)
Assessor-blinded
Computerized randomization system with 2x2 factorial design
Yes
Does empagliflozin, sustained-release carvedilol, or their combination improve right ventricular remodeling and reduce tricuspid regurgitation severity in patients with significant functional tricuspid regurgitation?
Mean Difference: 1.67 (95% CI -7.25–10.59)
Absolute Event Rate: -1.97% vs -3.64%
p-value: p=0.708
In patients with functional tricuspid regurgitation, empagliflozin significantly reduces tricuspid regurgitation severity but does not improve right ventricular remodeling.
Kim et al. (2026) conducted an RCT in Functional tricuspid regurgitation and stable heart failure (n=56). Empagliflozin vs. Placebo was evaluated on Change in right ventricular end-systolic volume index from baseline to 12 months (MD 1.67, 95% CI -7.25 to 10.59, p=0.708). Empagliflozin did not significantly reduce right ventricular end-systolic volume index compared with placebo (MD 1.67 mL/m2, p=0.708), although it significantly reduced secondary measures of tricuspid regurgitation severity.