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August 17, 2026Acta Biochimica et Biophysica SinicaOpen Access

Multicellular human iPSC-derived cardiac organoids successfully model key features of overnutrition, HF, and MI.

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Why the study?

Cardiovascular diseases remain leading causes of mortality, yet therapeutic development is hindered by a lack of adequate human-relevant models.

Population

Human iPSC-derived cardiac organoids incorporating cardiomyocytes, endothelial cells, fibroblasts, and macrophages

Design

Preclinical in vitro model development study

Key result

A multicellular human iPSC-derived cardiac organoid system with real-time calcium imaging successfully recapitulated key features of overnutrition, heart failure, and myocardial infarction.

Authors

VKVishnu Goutham KotaSGShachin Velur GanesanYHYin Love Ho

Discussion

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Overview

May advance human iPSC-based modeling of heart failure and MI; leaves open clinical translation pending validation.

Structured PICO

P
Population
human iPSC-derived cardiac organoids (multicellular, incorporating cardiomyocytes, endothelial cells, fibroblasts, and macrophages)
I
Intervention
Development of a multicellular human iPSC-derived cardiac organoid system with a genetically encoded calcium reporter and optimized oxygen-permeable culture
O
Outcome
Functional modeling of heart failure and myocardial infarction (contractility, calcium dynamics, and biomarker expression)surrogate

A novel multicellular human iPSC-derived cardiac organoid system provides a scalable and physiologically relevant platform for studying cardiac disease mechanisms, drug responses, and cardiotoxicity.

Cite This Study

Kota et al. (2026) studied Heart failure and myocardial infarction. Multicellular human iPSC-derived cardiac organoid system was evaluated. A multicellular human iPSC-derived cardiac organoid system with real-time calcium imaging successfully recapitulated key features of overnutrition, heart failure, and myocardial infarction.

synapsesocial.com/papers/6aa914bc39e8b1c7dca349cehttps://doi.org/10.3724/abbs.2026152
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