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January 1, 2016Journal of Leukocyte Biology

Editorial: Proton pathway paradox: Hv1 H+ channel sustains neutrophil Nox2 activity, yet suppresses HOCl formation

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Abstract

Discussion on the functional roles of Hv1 proton channels in activated neutrophils.

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Cite This Study

Robert A. Clark (2016) studied this question.

synapsesocial.com/papers/6aa9e725f92ab359461bf290 https://doi.org/10.1189/jlb.4ce0515-188rr

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Voltage-Gated Proton Channels and Other Proton Transfer PathwaysOriginal Research · 2003 · 737 citations
2Hv1 proton channels are required for high-level NADPH oxidase-dependent superoxide production during the phagocyte respiratory burstOriginal Research · 2009 · 267 citations
3Modeling the Reactions of Superoxide and Myeloperoxidase in the Neutrophil PhagosomeOriginal Research · 2006 · 636 citations
4The voltage-gated proton channel Hv1/VSOP inhibits neutrophil granule releaseOriginal Research · 2015 · 23 citations
5Identification of Thr29 as a Critical Phosphorylation Site That Activates the Human Proton Channel Hvcn1 in Leukocytespreclinical · 2009 · 64 citations