Key result
IV and oral potassium promptly resolves acute hypokalemic paralysis in a pregnant woman with gestational hypertension.
Case Report (n=1)
Intravenous potassium replacement promptly resolves acute hypokalemic paralysis in pregnancy.
May support potassium repletion for hypokalemic paralysis in gestational hypertension; single case leaves generalizability open.
Hypokalemic myopathy may range from numbness/weakness to complete paralysis. The aetiology may be congenital or acquired. It is characterized by acute muscular weakness with low levels of potassium (<3.5 meq/L). We present a case of 26-year-old multigravida at 36 weeks of gestation with gestational hypertension on treatment, who came with acute onset of pain, numbness and weakness of both legs which worsened following betamethasone injection. She was diagnosed to have Hypokalemic paralysis with potassium levels of 2.1 meq/L. The medical profile remitted promptly on intravenous potassium replacement. Pregnancy was continued till 37 weeks with oral potassium supplements, antihypertensives and regular monitoring of serum potassium levels. The pregnancy was terminated after 37 weeks in view of gestational hypertension. Postpartum period was uneventful, patient was discharged after two weeks when potassium levels and BP returned to normal.
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M Kulkarni (2014) conducted a case report in Hypokalemic paralysis (n=1). Intravenous and oral potassium replacement was evaluated on Symptom remission and normalization of potassium levels. Intravenous and oral potassium replacement promptly resolved acute hypokalemic paralysis (potassium 2.1 meq/L) in a 26-year-old pregnant woman with gestational hypertension.
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