Equils et al. ([1][1]) reported implication of simultaneous activation of Toll-like receptors (TLRs) on HIV replication. When spleen cells from HIV-1 transgenic mice were exposed to agonists of TLR2, TLR4, and TLR9, p24 production was induced. The agonist of TLR9 used in this study was bacterial CpG
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Agrawal et al. (2003) studied this question.
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