Key result
Very low-volume surgeons linked to ~109% higher in-hospital mortality after open abdominal aortic repair.
Why the study?
Little research has focused on very low-volume vascular surgery practice and its outcomes amid decreasing vascular surgery volume with endovascular adoption.
Does elective vascular surgery performed by very low-volume surgeons worsen in-hospital outcomes and increase healthcare resource use compared to higher-volume surgeons?
Cohort (n=77,677)
Yes
Does elective vascular surgery performed by very low-volume surgeons worsen in-hospital outcomes and increase healthcare resource use compared to higher-volume surgeons?
Answers are grounded in this paper and cite their sources.
Elective vascular surgeries (OAR and CEA) performed by very low-volume surgeons are associated with significantly worse clinical outcomes and higher healthcare resource utilization compared to those performed by higher-volume surgeons.
IMPORTANCE: Little research has focused on very low-volume surgery, especially in the context of decreasing vascular surgery volume with the adoption of endovascular procedures. OBJECTIVE: To investigate the existence and outcomes of open abdominal aortic aneurysm repair (OAR) and carotid endarterectomy (CEA) performed by very low-volume surgeons in New York. DESIGN, SETTINGS, AND PARTICIPANTS: This cohort study examined inpatient data of patients undergoing elective OAR or CEA from 2000 to 2014 from all New York hospitals. EXPOSURES: Surgeons who performed 1 or less designated procedure per year on average were considered very low volume, as opposed to higher-volume surgeons. MAIN OUTCOMES AND MEASURES: Temporal trends of the existence of very low-volume practice were evaluated. Hierarchical logistic regression was used to compare in-hospital outcomes and health care resource use between patients treated by very low-volume surgeons and higher-volume surgeons for both OAR and CEA, adjusting for patient, surgeon, and hospital characteristics. RESULTS: There were 8781 OAR procedures and 68 896 CEA procedures included in the study. The mean (SD) patient age was 71.7 (8.4) years for OAR and 71.5 (9.1) years for CEA. A total of 614 surgeons performed OAR and 1071 performed CEA in New York during the study period. Of these, 318 (51.8%) and 512 (47.8%), respectively, were very low-volume surgeons. Very low-volume surgeons were less likely to be vascular surgeons. The number and proportion of very low-volume surgeons decreased over years. Compared with patients treated by higher-volume surgeons, those treated by very low-volume surgeons were more likely to have higher in-hospital mortality (odds ratio [OR], 2.09; 95% CI, 1.41-3.08) following OAR and higher risks of postoperative myocardial infarction (OR, 1.83; 95% CI, 1.03-3.26) and stroke (OR, 1.78; 95% CI, 1.21-2.62) following CEA. Patients treated by very low-volume surgeons also had greater health care resource use following both surgeries, including prolonged length of stay (OR, 1.37; 95% CI, 1.11-1.70) following OAR as well as higher charges (OR, 1.28; 95% CI, 1.01-1.62) and increased 30-day readmission (OR, 1.30; 95% CI 1.04-1.62) following CEA. CONCLUSIONS AND RELEVANCE: The OAR and CEA procedures performed by very low-volume surgeons resulted in worse postoperative outcomes and greater lengths of stay. Although the percentage of very low-volume surgeons declined from 2000 to 2014, it remains concerning, given ready access to higher-volume surgeons. Future research is needed to understand the existence of this practice pattern in other surgical fields. Efforts to eliminate this practice pattern are warranted to ensure high-quality care for all patients.
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Mao et al. (2017) conducted a cohort in Abdominal aortic aneurysm and carotid artery disease (n=77,677). Very low-volume surgeons (≤1 procedure per year) vs. Higher-volume surgeons was evaluated on In-hospital mortality following OAR (OR 2.09, 95% CI 1.41-3.08). Very low-volume surgeons were associated with higher in-hospital mortality following OAR (OR 2.09; 95% CI 1.41-3.08) and higher risks of postoperative MI and stroke following CEA.
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