The author of this book finds little merit in the thesis that lipid deposition or mural thrombosis initiates the process by which coronary arteries may eventually become occluded. Even vessels with multiple lumens are not considered to contain organized and recanalized thrombi. He believes that some 35 distinct processes are involved and that the absence of lipid in a thickened intima is prima facie evidence that none was ever present. Osborn's views are ingenious and original but do not involve over-simplification. Novel interpretations require new nomenclature and this is richly supplied. A glossary might have been useful to relate such expressions as "triphasic parites," "angular activity," "necroliths," "feeders," and "dikes" to older terms in general usage. A unique system, that the author considers to be simple, is used to codify the lesions. These at first glance resemble tabulated stock market quotations in a Japanese financial journal. The expression 1, 0.5,
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Sigmund L. Wilens (1963) studied this question.