Why the study?
Some non-cardiac diseases can increase circulating troponin T levels, but its elevation in mechanically ventilated ALS patients with acute thoracic pain is not well characterized.
Case Report (n=1)
No
Isolated hs-cTnT elevation may reflect skeletal muscle in ALS; leaves open whether cardiac evaluation can be safely omitted.
Troponin T is a specific structural protein to the heart and its detection in blood is determined by cardiomyocyte damage. It has also been reported that some non-cardiac disease can increase the circulating level of troponin T. Here we reported a case of Amyotrophic Lateral Sclerosis (ALS) patient with acute thoracic pain and a longstanding increase of troponin t blood levels.
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Graziani et al. (2015) conducted a case report in Amyotrophic Lateral Sclerosis (n=1). Amyotrophic lateral sclerosis was evaluated on High-sensitivity cardiac troponin T (hs-cTnT) levels.
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