, TGFBR1 knockdown reduced the proliferation and migration of GC cells, altered epithelial-mesenchymal transition-associated protein expression and promoted apoptosis. Collectively, these findings define the cancer-type-specific clinical and biological relevance of TGFBR1 and support its prognostic importance in selected malignancies, particularly GC, while further mechanistic and prospective validation remains necessary.
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Zhao et al. (2026) studied this question.
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