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Surface antigens of the human malaria parasite Plasmodium falciparum are under intense immune pressure, and their loci are subject to positive selection ([ 1 ][1], [ 2 ][2]). P. falciparum shows substantial genetic variability, as seen in rapid emergence of drug resistance [e.g., 10 mutations in
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Tanabe et al. (2004) studied this question.
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