Key result
Ashman phenomenon identified as the cause of wide complex beats mimicking ventricular tachycardia in near-syncope.
Why the study?
Differentiating atrial from ventricular ectopy in patients with bradycardia and premature wide complex beats is clinically important but can be challenging.
Case Report (n=1)
Highlights the importance of careful ECG evaluation to correctly identify the Ashman Phenomenon (aberrantly conducted atrial ectopy) mimicking premature ventricular complexes.
The above electrocardiogram (ECG) was obtained in a patient with near syncope and bradycardia. The rhythm was initially interpreted electronically as sinus bradycardia, left ventricular hypertrophy with strain pattern, left atrial enlargement and premature ventricular complexes (PVCs). The heart rate as evident from the rhythm strip was approximately 51 beats per minute with a cycle length of 1154 ms. Two early coupled premature wide complex beats are noted (red arrows) with right bundle branch block morphology. Closer examination of these premature beats reveals non-sinus P waves preceding each beat and fused with a prior T wave (green arrows). The cycle length spanning the sinus p wave preceding the ectopic beat till the sinus beat following it is 1659 ms, less than twice the sinus cycle length, indicating a non-compensatory pause. Both of these observations are highly suggestive of an atrial origin of the ectopic beats which conducted with aberration due to a long-short cycle length causing refractoriness of the right bundle (Ashman Phenomenon).
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Parks et al. (2026) conducted a case report in Near syncope and bradycardia (n=1). Premature wide complex beats on the ECG of a patient with near syncope were identified as atrial ectopy with aberrant conduction due to a long-short cycle length (Ashman Phenomenon).
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