Key result
Myxomatous mitral valve prolapse alters mediators of intracellular interaction and endothelial function in young adults.
Why the study?
The study aimed to investigate selected mediators of intracellular interactions and endothelial function in patients with myxomatous mitral valve prolapse (MVP).
Observational (n=25)
Answers are grounded in this paper and cite their sources.
Small observational study in young myxomatous MVP patients should not change practice; leaves open endothelial mediator roles for prospective validation.
Aim. To study the selected mediators of intracellular interactions and endothelial function in patients with myxomatous mitral valve prolapse (MVP). Material and methods. In total, 25 patients with myxomatous MVP, aged 18–29 years, were examined. The comparison groups included patients with MVP but no echocardiographic signs of myxomatosis and healthy individuals. The blood levels of the following mediators were measured: interleukin 1
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Yagoda et al. (2013) conducted an observational in Myxomatous mitral valve prolapse (n=25). Myxomatous mitral valve prolapse vs. Patients with mitral valve prolapse without myxomatosis and healthy individuals was evaluated on Mediators of intracellular interactions (including interleukin 1) and endothelial function. Myxomatous mitral valve prolapse was evaluated for intracellular interaction mediators and endothelial function in 25 patients aged 18–29 years (results truncated in abstract).
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