Key result
Staged open repair and TEVAR yields 100% patency, but intentional LSA coverage triggers subclavian steal.
Why the study?
Contemporary management of chronic post-dissection aortic disease includes open, endovascular, and staged open-endovascular strategies, with treatment selection depending on anatomy, rupture risk, malperfusion, physiological reserve, and device availability.
Case Report (n=1)
No
This case highlights that staged segment-specific open infrarenal and thoracic endovascular repair can successfully treat complex heterogeneous post-dissection aortic aneurysms, but reinforces guideline recommendations for prophylactic left subclavian artery revascularization when zone 2 TEVAR follows prior abdominal aortic surgery.
Answers are grounded in this paper and cite their sources.
Supports staged hybrid repair in select complex post-dissection aneurysms; leaves open broader applicability and LSA strategies pending larger series.
Background: Contemporary management of chronic post-dissection aortic disease includes open repair, thoracic endovascular aortic repair (TEVAR) with or without adjunctive false-lumen occlusion, fenestrated/branched endovascular repair, and staged open-endovascular strategies. Selection depends on anatomy, rupture risk, malperfusion, physiological reserve, and device availability. Case presentation: and major cardiometabolic comorbidity presented with progressive abdominal pulsation. CTA demonstrated chronic Stanford type B/DeBakey III dissection with descending thoracic aneurysmal degeneration to 52.7 mm, a dominant symptomatic infrarenal aneurysmal component measuring approximately 11-12 cm, bilateral common iliac involvement, and residual paravisceral dissection without treatment-triggering enlargement or malperfusion. No Crawford extent was assigned because complete longitudinal centerline imaging was unavailable. Stage I comprised transperitoneal open aorto-bi-iliac reconstruction. After 118 days, elective morphology-driven zone 2 TEVAR was performed to cover the reported proximal entry region and reduce antegrade false-lumen inflow. Intentional left subclavian artery (LSA) coverage without prophylactic revascularization was followed by symptomatic subclavian steal requiring unplanned carotid-subclavian bypass. Results: No perioperative death, stroke, spinal cord ischemia, bowel ischemia, myocardial infarction, acute kidney injury, graft thrombosis, prosthetic infection, or access complication occurred. At latest follow-up, the patient was asymptomatic and all reconstructions were patent. Conclusion: This case illustrates competing priorities in segment-specific management of chronic post-dissection aortic disease. The unequivocal indication for urgent infrarenal repair contrasted with discretionary pre-emptive thoracic intervention below the conventional diameter threshold. The LSA-related complication emphasizes prophylactic LSA preservation/revascularization, spinal collateral planning, and lifelong surveillance of the untreated paravisceral segment after previous abdominal aortic reconstruction.
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Chinaliyev et al. (2026) conducted a case report in Chronic Stanford type B/DeBakey III post-dissection aortic disease with descending thoracic (52.7 mm) and giant infrarenal (11-12 cm) aneurysmal components (n=1). Staged open transperitoneal aorto-bi-iliac reconstruction followed by zone 2 TEVAR with intentional LSA coverage and subsequent carotid-subclavian bypass was evaluated on Perioperative major adverse events and graft patency at latest follow-up. Staged open aorto-bi-iliac repair and zone 2 TEVAR achieved 100% reconstruction patency without mortality, though intentional LSA coverage caused subclavian steal requiring carotid-subclavian bypass.
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