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Asthma can be divided into at least two distinct molecular phenotypes defined by degree of Th2 inflammation. Th2 cytokines are likely to be a relevant therapeutic target in only a subset of patients with asthma. Furthermore, current models do not adequately explain non-Th2-driven asthma, which represents a significant proportion of patients and responds poorly to current therapies.
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Woodruff et al. (Fri,) studied this question.
www.synapsesocial.com/papers/69df068f58b92af24d7a0d3c — DOI: https://doi.org/10.1164/rccm.200903-0392oc
Prescott G. Woodruff
Barmak Modrek
David F. Choy
American Journal of Respiratory and Critical Care Medicine
University of California, San Francisco
Pulmonary and Critical Care Associates
University of San Francisco
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