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MI is associated with tissue-specific activation of myocardial aldosterone synthesis. This increase is mediated primarily by cardiac angiotensin II via AT1-subtype receptor and may be involved in post-MI ventricular fibrosis and in control of tissue norepinephrine concentration.
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Jean‐Sébastien Silvestre
Christophe Heymes
Abdeslam Oubénaı̈ssa
Circulation
Inserm
Délégation Paris 7
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Silvestre et al. (Tue,) studied this question.
www.synapsesocial.com/papers/69af0aefe03f6d79082a8c73 — DOI: https://doi.org/10.1161/01.cir.99.20.2694
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