BDNF/TrkB signaling protects against myocardial infarction in rodents by regulating TRPC3/6 channels, representing a novel potential therapeutic candidate for MI.
BDNF/TrkB alleviated cardiac ischemic injury and inhibited cardiomyocytes apoptosis by regulating TRPC3/6 channels, which provides a novel potential therapeutic candidate for MI.
Hang et al. (Thu,) studied this question.