BDNF/TrkB signaling protects against myocardial infarction in rodents by regulating TRPC3/6 channels, representing a novel potential therapeutic candidate for MI.
Hypothesis-generating for BDNF/TrkB in ischemic injury; human studies required before clinical consideration.
BDNF/TrkB alleviated cardiac ischemic injury and inhibited cardiomyocytes apoptosis by regulating TRPC3/6 channels, which provides a novel potential therapeutic candidate for MI.
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Hang et al. (2015) studied this question.
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