Key points are not available for this paper at this time.
Anti-neutrophil cytoplasmic antibody (ANCA)–associated vasculitis (AAV) represents a group of systemic autoimmune small vessel vasculitis, leading to significant morbidity and mortality due to extensive organ damage (including kidney failure)1 and treatment toxicity to medications such as glucocorticoids.2 Thus, research focuses on developing treatment that minimize glucocorticoid use.
Zimmermann et al. (Sat,) studied this question.