September 5, 2024Open Access

Innate T‐cell‐derived IL‐17A/F protects from bleomycin‐induced acute lung injury but not bleomycin or adenoviral TGF‐β1‐induced lung fibrosis in mice

Key Points

Key points are not available for this paper at this time.

Abstract

The pathobiology of IL-17 in lung fibrogenesis is controversial. Here we examined the role of IL-17A/F in bleomycin (BLM) and adenoviral TGF-β1-induced lung fibrosis in mice. In both experimental models, WT and IL17af

Connected Papers

Building similarity graph...

Analyzing shared references across papers

Discussion

Authors

Marie Moog

Medizinische Hochschule Hannover

Melina Baltes

Medizinische Hochschule Hannover

Tina Röpke

Medizinische Hochschule Hannover

Journals

European Journal of Immunology

Actions

Institutions

McMaster University

Universität Hamburg

RWTH Aachen University

References and Citations

Connected Papers

Building similarity graph...

Analyzing shared references across papers

Innate T‐cell‐derived IL‐17A/F protects from bleomycin‐induced acute lung injury but not bleomycin or adenoviral TGF‐β1‐induced lung fibrosis in mice

Key Points

Abstract

Citation Network

Connected Papers

Discussion

Authors

Journals

Actions

Institutions

References and Citations

Citation Network

Connected Papers

Discussion

Cite this study