Figure S2 from PPARδ Orchestrates a Prometastatic Metabolic Response to Microenvironmental Cues in Pancreatic Cancer

Key Points

• No changes in CD133 expression or self-renewal were observed in pancreatic cancer cells after treatment.
• The treatment involved etomoxir and macrophage-conditioned medium, indicating potential metabolic interactions.
• The metabolic response to microenvironmental cues plays a significant role in pancreatic cancer progression.
• Findings suggest that PPARδ may orchestrate metabolic pathways in pancreatic cancer without altering stemness markers.