Neurodevelopmental conditions are characterized by differences in sensory processing, motor control, and autonomic regulation. While substantial empirical research exists on individual components such as fascia as a sensory organ, interoceptive processing differences, and mechanotransduction, these findings have not yet been integrated into a coherent model focused on specific anatomical zones. This paper proposes that chronic mechanical restriction within sensor-rich anatomical zones — including the extraocular muscles, tensor tympani and stapedius, nasal musculature, tongue and floor of mouth, pharyngeal and laryngeal structures, suboccipital complex, visceral fascia and diaphragm, and pelvic floor and perineum — can distort afferent signaling through both neural and fluid-mediated pathways. Drawing on existing empirical literature on fascia as a sensory organ, interoception in autism, mechanotransduction, and biotensegrity, this paper outlines a mechanistic framework in which sustained constriction in these high-density sensory interfaces may contribute to the persistence of sensory and regulatory differences. The hypothesis identifies a set of anatomical targets and proposes that systematic investigation using objective tension measurement technologies represents a necessary and feasible next step.
Hai Young Kim (Wed,) studied this question.
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