Muscle atrophy may be influenced by genes FOS and CCL2, indicating potential treatment targets.
FOS and CCL2 likely play a role in the molecular pathophysiology of muscle atrophy after spinal cord injury.
Analysis focused on gene expression and mechanisms involved in spinal cord injury-related muscle loss in rats.
Identifying these genes may enable better treatment strategies for muscle atrophy post-injury.
Abstract
FOS and CCL2 may be involved in the molecular pathophysiology of muscle atrophy after SCI, serving as potential targets for diagnosis or treatment of SCI-related muscle atrophy.