Inhibition of H3K14 lactylation promotes diabetic corneal nerve regeneration via Wnt1/β-catenin signaling

Key Points

• Inhibition of H3K14 lactylation significantly promotes diabetic corneal nerve regeneration.
• Nerve regeneration was linked to the activation of Wnt1 and β-catenin signaling pathways.
• Analysis focused on diabetic models exhibiting altered corneal nerve structures.
• Findings suggest potential therapeutic strategies to address nerve degeneration in diabetes.