The abstract describes a secondary analysis to determine if reduced average skin sympathetic nerve activity during sleep is associated with ST elevation in hPOTS, but reports no results.
Observational
Is reduced average skin sympathetic nerve activity (aSKNA) during sleep associated with ST elevation in ambulatory participants with hPOTS treated with clonidine?
This secondary analysis investigates the relationship between reduced nocturnal skin sympathetic nerve activity and ST elevation in patients with hyperadrenergic POTS.
Hyperadrenergic postural orthostatic tachycardia syndrome (hPOTS) is a subgroup of POTS with prominent sympathetic activation symptoms. We recently recorded skin sympathetic nerve activity (SKNA) in ambulatory participants with hPOTS at baseline and after being treated with clonidine.1 We performed a secondary analysis to determine if reduced average SKNA (aSKNA) during sleep was associated with ST elevation.
Rao et al. (2026) conducted an observational in Hyperadrenergic postural orthostatic tachycardia syndrome (hPOTS). Reduced average skin sympathetic nerve activity (aSKNA) during sleep was evaluated on ST elevation. The abstract describes a secondary analysis to determine if reduced average skin sympathetic nerve activity during sleep is associated with ST elevation in hPOTS, but reports no results.
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