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Diet-induced atherosclerosis in macaque monkeys was suppressed by anticalcifying agents without changing abnormal levels of blood cholesterol and lipoprotein. The agents included inhibitors of arterial calcium deposition (diphosphonates) and a calcium ion antagonist (lanthanum). The study suggests that regulation of calcium flux and extracellular deposition in arteries may offer new principles of treatment for cardiovascular disease.
Kramsch et al. (Fri,) studied this question.