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Diet-induced atherosclerosis in macaque monkeys was suppressed by anticalcifying agents without changing abnormal levels of blood cholesterol and lipoprotein. The agents included inhibitors of arterial calcium deposition (diphosphonates) and a calcium ion antagonist (lanthanum). The study suggests that regulation of calcium flux and extracellular deposition in arteries may offer new principles of treatment for cardiovascular disease.
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Kramsch et al. (1981) studied this question.
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