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September 10, 2025

Mechanisms Underlying Hazardous Alcohol Use After Mild Traumatic Brain Injury.

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Authors

MPMakenzie PatarinoJSJeffrey N SandersASAbigail G. Schindler

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Overview

This review uncovers biological mechanisms connecting hazardous alcohol use with mild traumatic brain injury, suggesting preventative strategies.

Key Points

  • Alcohol use disorder significantly complicates the outcomes for individuals after mild traumatic brain injury.
  • Biological mechanisms like inflammation and neuroendocrine alterations are associated with hazardous alcohol use post-mTBI.
  • This review employs a literature assessment approach to evaluate studies on alcohol use following mild traumatic brain injury.
  • Understanding these mechanisms may help develop effective treatment and prevention strategies for at-risk individuals.

Cite This Study

Patarino et al. (2025) studied this question.

synapsesocial.com/papers/68c199e89b7b07f3a061b5fchttps://doi.org/10.35946/arcr.v45.1.09
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Traumatic brain injury and alcohol: A narrative review of the role of mitochondrial dysfunction and ferroptosis in neurocognitive outcomes2026
  2. 2Effects of Alcohol Use Status on Clinical Characteristics and Outcomes in Patients With Traumatic Brain Injury2026
  3. 3Traumatic Brain Injury Exacerbates Alcohol Consumption and Neuroinflammation with Decline in Cognition and Cholinergic Activity2024 · 2 citations
  4. 4Investigating the Behavioral and Biological Effects of Chronic Alcohol Use in a Mouse Model of Blast-Induced Mild Traumatic Brain Injury2024
  5. 5Alcohol-Mediated Cognitive Impairment Following Traumatic Brain Injury: Evidence for Cortical Mitochondrial Dysfunction2026