Bidirectional Mendelian randomization reveals causal pathways in aneurysmal subarachnoid hemorrhage, highlighting immune influences.
Background The causal interplay between inflammatory mediators and immune cell phenotypes in aneurysmal subarachnoid hemorrhage (aSAH) remains undefined. We investigated whether immune surface antigens influence rupture risk through inflammatory protein mediation.
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Yan et al. (2025) studied this question.
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