Observational study reveals altered inflammatory cytokine signatures in pediatric long COVID patients, suggesting persistent immune dysregulation following SARS-CoV-2 infection.
We provide preliminary evidence that, also in children, Long coronavirus disease (COVID) may be characterized by a proinflammatory signature. Ten Long COVID patients, 7 convalescent subjects after COVID infection and 4 healthy controls were enrolled. When adjusted for sex, children with long COVID had statistically significant differences in the levels of Flt3L, CD5, uPA, CCL23, CD40 and TGFα. When adjusted for age, CCL23 levels remained statistically significant.
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Buonsenso et al. (2024) studied this question.
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