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September 16, 2024

Figure S6 from β-catenin drives butyrophilin-like molecule loss and γδ T-cell exclusion in colon cancer

Toshiyasu Suzuki Anna Kilbey Nuria Casa-Rodríguez Amy Lawlor Αναστασία Γεωργακοπούλου Hannah Hayman Kyi Lai Yin Swe Anna Nordin Claudio Cantù Pierre Vantourout Rachel A. Ridgway Ryan M. Byrne Lei Chen Michael P. Verzi David M. Gay Ester Gil Vázquez Hayley L. Belnoue-Davis Kathryn Gilroy Anne Helene Køstner Christian Kersten Chanitra Thuwajit Ditte K. Andersen Robert Wiesheu Anett Jandke Karen Blyth Antonia K. Roseweir Simon J. Leedham Philip D. Dunne Joanne Edwards Adrian Hayday Owen J. Sansom Seth B. Coffelt

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Abstract

Identification of a WNT responsive element (WRE) in the HNF4G genomic locus.

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Cite This Study

Suzuki et al. (2024) studied this question.

synapsesocial.com/papers/68e587eeb6db643587523da2 https://doi.org/10.1158/2326-6066.27026735.v1

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Figure S1 from β-catenin drives butyrophilin-like molecule loss and γδ T-cell exclusion in colon cancer2024
2Figure S5 from β-catenin drives butyrophilin-like molecule loss and γδ T-cell exclusion in colon cancer2024
3Figure S2 from β-catenin drives butyrophilin-like molecule loss and γδ T-cell exclusion in colon cancer2024
4Figure S4 from β-catenin drives butyrophilin-like molecule loss and γδ T-cell exclusion in colon cancer2024
5Figure S3 from β-catenin drives butyrophilin-like molecule loss and γδ T-cell exclusion in colon cancer2024