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August 16, 2024Environment & HealthOpen Access

Effects on Synaptic Plasticity Markers in Fetal Mice and HT22 Neurons upon F-53B Exposure: The Role of PKA Cytoplasmic Retention

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SLShen-Pan LiHZHui-Xian ZengSQShuang-Jian Qin

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Li et al. (2024) studied this question.

synapsesocial.com/papers/68e5be81b6db6435875568f3https://doi.org/10.1021/envhealth.4c00098
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Early F-53B Exposure Induces Autism Spectrum Disorder-like Hypomyelination and Oligodendrocytes-Derived Exosomal Protein-Dependent Axonal Energy Imbalance2026 · 3 citations
  2. 2F-53B-induced placental vascular endothelial dysfunction leads to intrauterine growth retardation of fetal mice2025
  3. 3Placental inflammatory injury induced by chlorinated polyfluorinated ether sulfonate (F-53B) through NLRP3 inflammasome activation2024 · 11 citations
  4. 4Maternal F-53B exposure during pregnancy and lactation affects bone growth and development in male offspring2024 · 23 citations
  5. 5Integrating Adverse Outcome Pathway–Bayesian Network and Physiologically Based Pharmacokinetic Modeling for Quantitative Prediction of Developmental Neurotoxicity of F-53B under Human-Relevant Exposure Scenarios2026 · 1 citations