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July 9, 2024

Supplementary Figure 4 from KRAS Promotes GLI2-Dependent Transcription during Pancreatic Carcinogenesis

Ashley N. Sigafoos Ezequiel J. Tolosa Ryan M. Carr Maite G. Fernández‐Barrena Luciana L. Almada David R. Pease Tara L. Hogenson Glancis L. Raja Arul Fatemeh Mousavi Sandhya Sen Renzo E. Vera David L. Marks Luis F. Flores Kayla C. LaRue-Nolan Chen Wu William R. Bamlet Anne M. Vrabel Hugues Sicotte Erin L. Schenk Thomas C. Smyrk Lizhi Zhang Kari G. Rabe Ann L. Oberg Peter G. Zaphiropoulos Éric Chevet Rondell P. Graham Catherine E. Hagen Marina Pasca di Magliano Sherine F. Elsawa Christopher L. Pin Junhao Mao Robert R. McWilliams Martín E. Fernández-Zapico

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Abstract

Supplementary Figure 4 shows the validation of Kras signaling activation and chronic pancreatitis phenotypic examples in KC and KCRG mice.

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Cite This Study

Sigafoos et al. (2024) studied this question.

synapsesocial.com/papers/68e60f5cb6db6435875a1fb1 https://doi.org/10.1158/2767-9764.26213834

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure 4 from KRAS Promotes GLI2-Dependent Transcription during Pancreatic Carcinogenesis2024
2Supplementary Figure 4 from KRAS Promotes GLI2-Dependent Transcription during Pancreatic Carcinogenesis2024
3Supplementary Figure 4 from KRAS Promotes GLI2-Dependent Transcription during Pancreatic Carcinogenesis2024
4Supplementary Figure 5 from KRAS Promotes GLI2-Dependent Transcription during Pancreatic Carcinogenesis2024
5Supplementary Figure 5 from KRAS Promotes GLI2-Dependent Transcription during Pancreatic Carcinogenesis2024