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June 4, 2024Open Access

Supplementary Figure 6 from SPARC Stabilizes ApoE to Induce Cholesterol-Dependent Invasion and Sorafenib Resistance in Hepatocellular Carcinoma

Shan Wan Quan-Yao He Yun Yang Feng Liu Xue Zhang Xin Guo Hui Niu Yi Wang Yi-Xuan Liu Wen-Long Ye Xiuming Li Xue-Mei ZhuanSun Pu Sun Xiao‐Shun He Guang Hu Kai Breuhahn Hua Zhao Guo-Qiang Wu Hua Wu

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Abstract

SPARC is upregulated and maintains the cholesterol-dependent EMT phenotype in sorafenib-resistant HCC cells.

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Cite This Study

Wan et al. (2024) studied this question.

synapsesocial.com/papers/68e664bab6db6435875f1354 https://doi.org/10.1158/0008-5472.25964453.v1

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplementary Figure 6 from SPARC Stabilizes ApoE to Induce Cholesterol-Dependent Invasion and Sorafenib Resistance in Hepatocellular Carcinoma2024
2Supplementary Figure 2 from SPARC Stabilizes ApoE to Induce Cholesterol-Dependent Invasion and Sorafenib Resistance in Hepatocellular Carcinoma2024
3Supplementary Figure 2 from SPARC Stabilizes ApoE to Induce Cholesterol-Dependent Invasion and Sorafenib Resistance in Hepatocellular Carcinoma2024
4Supplementary Figure 5 from SPARC Stabilizes ApoE to Induce Cholesterol-Dependent Invasion and Sorafenib Resistance in Hepatocellular Carcinoma2024
5Supplementary Figure 5 from SPARC Stabilizes ApoE to Induce Cholesterol-Dependent Invasion and Sorafenib Resistance in Hepatocellular Carcinoma2024