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June 1, 2024Clinical and Translational MedicineOpen Access

Inhibiting the cGAS‐STING pathway in myeloid cells effectively improves myocardial healing related to TET2 deficiency‐induced DNA damage response

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Authors

YDYaling DouYZYan ZhangLRLogan Rivera

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Cite This Study

Dou et al. (2024) studied this question.

synapsesocial.com/papers/68e672d9b6db6435875fd283https://doi.org/10.1002/ctm2.1741
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Accelerated adverse cardiac remodeling in a model of TET2 mutation-driven clonal hematopoiesis is ameliorated by IFNa treatment by modulating the monocyte response2025
  2. 2Sting reestablishes mitochondrial homeostasis in heart failure2025
  3. 3Small molecule STING inhibition improves myocardial infarction remodeling2021 · 76 citations
  4. 4Activation of the cGAS-STING pathway in cardiomyocytes with short telomeres opens new therapeutic avenues to treat heart failure2025
  5. 5The Cardioprotective Role of Neutrophil-Specific STING in Myocardial Ischemia/Reperfusion Injury2024