Experimental mouse study shows that loss of ADAM29 accelerates wound healing without altering viability or fertility, suggesting a regulatory role in tissue repair.
ADAM29 (a disintegrin and metalloprotease domain 29) is a member of the membrane-anchored ADAM family of proteins, which is highly expressed in testis and may mediate different physiological and pathological processes. Although the functions of many ADAM family members have been well characterized, the biological relevance of ADAM29 has remained largely unknown. Here, we report the generation of an
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Campos-Iglesias et al. (2024) studied this question.
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