Managing scar-related ventricular tachycardia (VT) demands a nuanced understanding of the interplay between structural damage and electrophysiological function. Despite the individual contributions of late gadolinium enhancement cardiac magnetic resonance (LGE-CMR) and electrocardiographic imaging (ECGI) in mapping fibrotic tissue and electrical activity, their integration remains unexplored. This gap hinders a comprehensive, noninvasive evaluation of the proarrhythmic substrate, essential for effective VT management.
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Hernández‐Romero et al. (2024) studied this question.
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