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May 1, 2024Physiology

IF1 Knockout Protects Against Doxorubicin-Induced Cardiac Dysfunction by Improving Cardiomyocyte Energetics

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Authors

PMParnia MobasheranMDMeagan DonovanQYQinglin Yang

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Cite This Study

Mobasheran et al. (2024) studied this question.

synapsesocial.com/papers/68e6c6ffb6db643587645d91https://doi.org/10.1152/physiol.2024.39.s1.863
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1ATP Synthase Inhibitory Factor-1 Deficiency Attenuates Doxorubicin Cardiotoxicity by Preserving Mitochondrial Structure and Function2026
  2. 2Abstract Fri080: Kynurenic acid mitigates doxorubicin-cardiomyopathy via improving mitochondrial function and silencing ATP synthase inhibitory subunit 12025
  3. 3Doxorubicin‐induced cardiotoxicity involves IFNγ‐mediated metabolic reprogramming in cardiomyocytes2018 · 57 citations
  4. 4High-intensity Interval Training Protectively Modulates Doxorubicin-Induced Changes in Cardiac Mitochondrial Dynamics and Autophagy Process2025 · 1 citations
  5. 5Intermittent Fasting Attenuates Apoptosis, Modulates Autophagy and Preserve Telocytes in Doxorubicin Induced Cardiotoxicity in Albino Rats: A Histological Study2020 · 2 citations