IBS is one of the most well-known functional bowel disorders, with more than 10% of the global adult population reporting symptoms consistent with the illness in population-based studies. In standard clinical practice, an IBS diagnosis is obtained based on common symptoms.3 Investigations are frequently limited to a chosen panel of tests that aid in the exclusion of established organic disorders with comparable symptoms, such as inflammatory bowel disease or celiac disease. The actual etiology of IBS is unknown, however anomalies in the gut-brain axis, increased gastrointestinal sensitivity, and dysregulation of gut motility and sensation are thought to have a role. There is evidence that people with IBS are more likely to have fibromyalgia, chronic fatigue syndrome, and depression than the general population. Furthermore, autoimmune illnesses such as rheumatoid arthritis and inflammatory bowel disease are more commonly associated with IBS. The precise processes causing this relationship are unknown, however it is thought that shared genetic, environmental, and neurological variables may contribute to both IBS and other disorders. Understanding this link is critical for effectively managing and treating both IBS and related comorbidities, potentially leading to more complete care plans for affected individuals. Diet has a substantial impact on susceptibility to Irritable Bowel Syndrome (IBS), a common gastrointestinal condition marked by stomach pain, bloating, and changes in bowel movements. Certain dietary components have been identified as triggers for IBS symptoms, which exacerbates the condition in sensitive individuals. Diet does not directly cause IBS, but it can have a substantial impact on illness susceptibility and symptom severity. Adopting a balanced diet high in soluble fiber, probiotics, and nutrient-dense foods while avoiding recognized trigger foods may help to control IBS symptoms and improve overall gastrointestinal health.
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Rani et al. (2024) studied this question.
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