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April 3, 2024Open Access

Supplemental Figure 3 from Interaction between Continuous Pack-Years Smoked and Polygenic Risk Score on Lung Cancer Risk: Prospective Results from the Framingham Heart Study

Meredith S. Duncan Héctor Díaz-Zabala James Jaworski Hilary A. Tindle Robert A. Greevy Loren Lipworth Rayjean J. Hung Matthew S. Freiberg Melinda C. Aldrich

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Abstract

Supplemental Figure 3: PRS Model fit by p-value threshold. High resolution plots displaying the -log10(p-value) of the association between lung cancer and the PRS in the FHS sample (y-axis) versus the threshold below which all SNPs with p-values for their individual association with lung cancer in the base sample (OncoArray Consortium) were included (x-axis).

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Cite This Study

Duncan et al. (2024) studied this question.

synapsesocial.com/papers/68e709f8b6db64358768398c https://doi.org/10.1158/1055-9965.25532565.v1

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Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

1Supplemental Figure 3 from Interaction between Continuous Pack-Years Smoked and Polygenic Risk Score on Lung Cancer Risk: Prospective Results from the Framingham Heart Study2024
2Data from Interaction between Continuous Pack-Years Smoked and Polygenic Risk Score on Lung Cancer Risk: Prospective Results from the Framingham Heart Study2024
3Supplemental Figure 2 from Interaction between Continuous Pack-Years Smoked and Polygenic Risk Score on Lung Cancer Risk: Prospective Results from the Framingham Heart Study2024
4Supplemental Figure 2 from Interaction between Continuous Pack-Years Smoked and Polygenic Risk Score on Lung Cancer Risk: Prospective Results from the Framingham Heart Study2024
5Supplemental Figure 4 from Interaction between Continuous Pack-Years Smoked and Polygenic Risk Score on Lung Cancer Risk: Prospective Results from the Framingham Heart Study2024