Oxalate nephropathy (OXN) is a frequent and underestimated cause of kidney failure in patients undergoing gastric bypass surgery (GBS) and is characterized by hyperoxaluria with massive deposition of calcium oxalate crystals in the renal parenchyma resulting in tubular damage with subsequent interstitial fibrosis. In these patients, hyperoxaluria occurs primarily as a result of increased enteric absorption. It is important to make an early diagnosis and treatment, since once the process of OXN has begun, the evolution towards end stage renal disease (ESRD) is very frequent and can develop quickly.
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Hernandez et al. (2024) studied this question.
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