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March 22, 2024Cancer Research

Abstract 1791: Metabolic rewiring promotes metastatic potential upon glutamine deprivation in STK11 null KRAS-driven lung adenocarcinoma

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Authors

SPShannon M. PriorLSLogan SandsSLSean M. Lenahan

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Prior et al. (2024) studied this question.

synapsesocial.com/papers/68e72cd9b6db6435876a63cahttps://doi.org/10.1158/1538-7445.am2024-1791
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Abstract 6107: Activation of NF-κB signaling drives pro-metastatic programs in STK11-deficient lung adenocarcinoma2026
  2. 2Abstract 2772: Mechanisms linking STK11 loss with metastatic potential in KRAS-mutated lung adenocarcinoma2024
  3. 3Abstract 6104: STK11 loss enhances stress-adaptive programs supporting shear resilience in circulating tumor cells from KRAS-driven lung adenocarcinoma2026
  4. 4Abstract 7366: Loss of STK11 suppresses lipid metabolism to attenuate KRAS-induced immunogenicity in patients with non-small cell lung cancer2024
  5. 5Abstract 1114: Evaluating strategies to harness metabolic vulnerabilities in cervical2024