Cardiac arrest (CA) remains the leading cause of death in patients with heart failure (HF). Adverse remodeling and myocardial fibrosis form an ideal substrate for the generation and propagation of fatal ventricular arrythmias. Consequently, anti-remodeling guideline directed medical therapies (GDMT) and widespread utilization of implantable cardioverter-defibrillators (ICDs) have helped decreased the burden of cardiac arrest in this population beginning in the early 2000s.1 However, relatively little is known about more recent trends in CA associated with HF.
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Jain et al. (2024) studied this question.
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