Focal atrial tachycardia (AT) accounts for up to 10% of supraventricular tachycardias, however despite this low incidence, their management presents difficulties due to limited response to medical management including DCCV, and a predisposition to initiate other atrial arrhythmias 1. ATs may result from micro-re-entry, triggered activity or enhanced automaticity and typically arise from characteristic locations in the atria, where the anatomic configuration may predispose to ectopic impulse generation and propagation 2.
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Amin et al. (2024) studied this question.
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