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March 1, 2024Journal of Biological ChemistryOpen Access

Abstract 2117 Crosstalk between nutrient sensor proteins, O-GlcNAc Transferase and mTORC1, is key to regulation of pancreatic β-cell mass and function

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Authors

SJSeokwon JoEAEmilyn U. Alejandro

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Cite This Study

Jo et al. (2024) studied this question.

synapsesocial.com/papers/68e76a1eb6db6435876dfaaahttps://doi.org/10.1016/j.jbc.2024.106315
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Nutrient sensor O-GlcNAc transferase maintains pancreatic beta-cell identity, in part, through positive regulation of pancreatic duodenal homeobox 12026
  2. 2Abstract 2180 O-GlcNAc Regulation of Pancreatic β-cell Identity through Transcription Factor Pdx12024
  3. 3Constitutive mTORC1 Activation in Skeletal Muscle Increases Inflammation but is not Sufficient to Impair Glucose Tolerance2026
  4. 4Critical roles for the TSC-mTOR pathway in β-cell function2009 · 101 citations
  5. 5High Potassium Suppresses Glycosuria and Gluconeogenesis in Tubule-specific mTORC2 Knockout Mice2024