Renal dysfunction due to hypercalcemia typically manifests with impairment in the ability to concentrate urine, decreased glomerular filtration rate (GFR), natriuresis, nephrocalcinosis and diabetes insipidus. Acute Tubular Necrosis (ATN) is a rare manifestation of severe hypercalcemia (defined as serum calcium greater than 20 mg/dl). We present a case of severe hypercalcemia resulting from Milk-Alkali Syndrome caused by chronic ingestion of calcium-based antacids (TUMS) resulting in Acute Kidney Injury (AKI) from ATN.
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Pandey et al. (2024) studied this question.
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