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February 21, 2024

Targeting ACSS2 activity suspends the formation of chemoresistance through suppressed histone H3 acetylation in human breast cancer

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Authors

XSXiaoxi ShuiLTLiying TianYZYuepeng Zhou

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Cite This Study

Shui et al. (2024) studied this question.

synapsesocial.com/papers/68e7845cb6db6435876f727bhttps://doi.org/10.21203/rs.3.rs-3964435/v1
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Acetyl-CoA Synthetase 2 Promotes Acetate Utilization and Maintains Cancer Cell Growth under Metabolic Stress2015 · 837 citations
  2. 2Fueling Disease: ACSS2 in Obesity‐Associated Metabolic Disorders and Cancer Progression2026 · 1 citations
  3. 3HIF-2α expression and metabolic signaling require ACSS2 in clear cell renal cell carcinoma2024 · 3 citations
  4. 4Abstract 1207 Interaction of metabolic and epigenetic control in colon cancer2024
  5. 5Abstract B066: Targeting ACSS2 promotes ferroptosis and immune cell infiltration in breast cancer brain metastases2026