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February 14, 2024Blood Cancer DiscoveryOpen Access

Human ASXL1-Mutant Hematopoiesis Is Driven by a Truncated Protein Associated with Aberrant Deubiquitination of H2AK119

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Authors

TKThomas KöhnkeKNKevin A. NunoCACatherine Alder

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Cite This Study

Köhnke et al. (2024) studied this question.

synapsesocial.com/papers/68e792b5b6db643587703792https://doi.org/10.1158/2643-3230.bcd-23-0235
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Also Consider

Synapse has enriched 5 closely related papers on similar clinical questions. Consider them for comparative context:

  1. 1Data from Human ASXL1-Mutant Hematopoiesis Is Driven by a Truncated Protein Associated with Aberrant Deubiquitination of H2AK1192024
  2. 2Supplementary Figures S1 - S11 from Human ASXL1-Mutant Hematopoiesis Is Driven by a Truncated Protein Associated with Aberrant Deubiquitination of H2AK1192024
  3. 3Supplementary Figures S1 - S11 from Human ASXL1-Mutant Hematopoiesis Is Driven by a Truncated Protein Associated with Aberrant Deubiquitination of H2AK1192024
  4. 4CH-related Mutant ASXL1 Promotes Atherosclerosis in Mice via Dysregulated Innate Immunity2024
  5. 5SMARCC1 loss impairs differentiation and enhances self-renewal in ASXL1-mutant hematopoietic cells2025